Tag Archives: MCTs

The Power of Being Thin Is Found By Eating Fat

The Power of Being Thin Is Found By Eating Fat

Most everybody wants to be thin simply to
look good,fat-thin-people-13593846 but the advantages of being thin go a lot further than just looking good. Being thin is not only highly beneficial for your looks but it’s crucial for your health and even more important for your brain’s health. Did you know that the fatter you are, the smaller your brain is? It’s true. That is directly from Dr Perlmutter’s book Grain Brain. Conversely, the thinner you are, the bigger your brain is. Don’t believe me? Look at the research studies and what Dr Perlmutter says in Grain Brain: 

“The dots connecting excessive body fat, obesity, and brain dysfunction are not hard to follow given the information you’ve already learned in this book. Excessive body fat increases not only insulin resistance, but also the production of inflammatory chemicals that play directly into brain degeneration. For this very reason, waist circumference is often a measurement of “health,” as it predicts future health challenges and mortality; the higher your waist circumference, the higher your risk for disease and death.”

danger-obesity-grim-reaper-touches-shoulder-happy-overweight-black-woman-big-cupcake-vector-illustration-health-41031554“It’s well documented that visceral fat is uniquely capable of triggering hormonal actions.  This, in turn, keeps the cascade of of negative effects from visceral fat going. In addition, visceral fat does more than just generate inflammation down the road through a chain of biological events; visceral fat itself becomes inflamed. This kind of fat houses tribes of inflammatory white blood cells. In fact, the hormonal and inflammatory molecules produced by visceral fat get dumped directly into the liver, which, as you can imagine, responds with another round of ammunition (i.e., inflammatory reactions and hormone-disrupting substances). Long story short: More than merely a predator lurking behind a tree, it is an enemy that is armed and dangerous. The number of health conditions now linked to visceral fat is tremendous, from the obvious ones such as obesity and metabolic syndrome to the not-so-obvious—cancer, autoimmune disorders, and brain disease.”

I copied and pasted the information above from Grain Brain for a reason. Obesity is a danger to more than just your body, by filling it with inflammation, it’s shrinking your brain by using these same process that creates plaque. I will show you exactly how obesity shrinks your brain and on the other hand, I’ll show you exactly how being thin can help your brain to grow in size. It all boils down to consumption of carbohydrates, mostly the high starchy carbs that you find in all pastries and breads, pastas, cereals, snack chips and crackers and some vegetables.

According to  Donald W. Miller, Jr., MD, Carbohydrates are the primary cause of weight gain, not fats. (Animals raised for food are fattened with carbohydrates.)” He goes on to say that eating fat is not only healthier than eating carbohydrates, it makes you thinner. “We found that the people who ate the most cholesterol, ate the most saturated fat, ate the most calories, weighed the least and were the most physically active” (Arch Int Med 1992;152:1271—2). It’s true,  I know from experience that eating fat makes you thin. It’s time for a new news alert;

Eating Fat Makes You Thin

Studies have shown that getting back to what our original metabolism likes for a diet and what our bodies are meant to digest means getting back to diet high in fats and low in carbohydrates. Low Carb diets date back to 1923 when the ketogenic diet was first created to help control epileptic seizures in infants. Dr Atkins came out with his low carb diet in 1958,  but it really got its boost when the Paleo diet came out early this century and with Dr Perlmutter’s recommendation for a ketogenic diet for optimal brain growth.

Wikipedia suggests, “we need to evaluate the low-carbohydrate diets over much longer periods of time, controlled studies as long as two years and survey studies as long as two decades.[7][13][14][15]” 

Dr Atkins was the first to promote a low carb diet as early as 1958, yet it seems that the carbohydrate addiction complex had already started its devious work in addicting our society to the ravages of the Wheat Belly saga. Too many members of our congress were sold on the notion that it is better to restrict our consumption fats, thinking that’s what was causing all the problem with obesity and diabetes. In all actuality, it is carbs that cause the fat that causes obesity and diabetes, not fat at all. You can find out how that happens in Carbs, The New Death Sentence. (I have to wonder who persuaded them to come to these conclusions, the grain industry?)

It’s all a matter of how they are digested. To digest carbohydrates, your body has to turn them into fat. This is because your body can’t run on glucose. It runs on fat. The studies showing this include,  Iris Shai, R.D., Ph.D. (July 2008), “Weight Loss with a Low-Carbohydrate, Mediterranean, or Low-Fat Diet.” and New England Journal of Medicine 359 (3): 229–41. doi:10.1056/NEJMoa0708681.PMID 18635428, Low-carbohydrate-Diet Score and the Risk of Coronary Heart Disease an Omen, from The New England Journal of Medicineand the two others listed above ([14][15]). What this means is that when you eat carbohydrates, your body can’t use that as food because it burns fat.

When you eat fat, your body doesn’t have to convert that into anything else, so it can use it. Fats are digested in your small intestine unlike carbs that are digested cellularly with the help of insulin. That means that the glucose that carbs break down to, have to float around in your blood stream until they can enter a cell to be used as glycogen. This is where the problem begins. Anyone who’s been on a diet of carbohydrates for any amount of time has enough glycogen built up in their systems that they don’t need anymore, so the glucose turns into fat to be stored for future use.

The first place your body stores this fat is around your mid section, hence its name, belly fat or visceral fat. This is a dangerous fat to have in your body as this is where diabetes starts, along with a host of cancers and CVDs or heart diseases and most every kind of dementia, including Alzheimer’s Disease, Parkinson’s Disease and Huntington’s Disease.

Human biology hasn’t changed evolutionarily enough to allow humans to continue to eat carbohydrates in the massive amounts that everyone everywhere is eating them. The Paleo Diet  is a recent addition to the low carb diet choice. The ketogenic diet is the ultimate in a low carb diet and has already shown numerous benefits for better health. It’s the recommended diet for Celiac Disease since Celiac Disease is caused by the gluten that’s found in wheat, barley and rye and a few other grains. It’s also the oldest low carb diet, first designed in 1923, to help control seizures. The diet fell out of use when seizure medicines became more prevalent.

It turns out that a ketogenic diet is the healthiest diet that any human can eat and it brings with it, the most natural form of weight loss, possible. It goes back to the way our bodies have  metabolized food for the last 100,000 years. Simply because this diet is based on fat and not carbs, the diet provides much more efficient fuel for our bodies to use. A carbohydrate diet requires refueling every two hours or so and they has a tendency to gum up your body. It does it by creating plaque. That gets into to glycation of proteins and LDL cholesterol, which you can read about in Carbs, How They Cause AGEs

This plaque build up is the foundation of 75% of the deadliest and costliest diseases, known to man, ranging from breast cancer  to Atherosclerosis to 99% of all dementias, making carbohydrates some of the deadliest food that any human can eat. It’s not that this food just makes us fat, it kills us slowly and expensively, with an arm long list of  disorders. For this one reason alone, the power of being thin cannot be overspoken.

Studies have also shown the simple practice of calorie restriction to have multiple beneficial effects for the body, such as extended life. It’s amazing what just going hungry, can do for your body. It not only ramps up your immune system by boosting your anti-oxidants exponentially, it actually helps your brain grow, through a little protein known as BDNF, brain derived neurotrophic factor. This is what makes your brain grow and it doesn’t happen in obese people. This is part of the power of being thin.

The Power of MCTs and Coconut Oil

Calorie restriction on a carbohydrate diet is next to impossible. Yet I do it every day, quite easily and comfortably, while on my MCT ketogenic diet. MCT ketogenic diet is, in my estimation, the easiest low carbohydrate diet to get adjusted to. MCTs (Medium Chain Triglycerides) work differently in your body than LCTs (Long Chain Triglycerides). MCTs are a good way to actually lower your cholesterol because they build up the HDL cholesterol. Coconut oil and Palm kernel oil are optimal for this, as they contain lauric acid  and lauric acid is the foundation of HDL cholesterol, the good cholesterol.

Although curbing your carbs is the best way to lower your LDL cholesterol, adding coconut oil and other saturated fats to your diet will help curb your appetite for carbs, which in turn will cut down your LDL cholesterol and at the same time build up your HDL cholesterol.

Going back to what Dr Miller had included in his paper, “calorie restriction prolongs life as well as helps to make your brain grow.” This is the true power of being thin. It comes easiest from being on a high fat low carb diet.

What kind of fats then, do we need to eat, to be thinner? I mentioned before, MCTs. Medium chain triglycerides are the best, along with olive oil and avocado oil, palm kernel oil is also an excellent source of MCTs but coconut oil is by far the best MCT, in my opinion.

Coconut oil and palm Kernel oil have lauric acid in their chains of triglycerides, which is the foundation of HDL cholesterol. This means that a diet high in coconut oil or palm kernel oil MCTs helps build up HDL cholesterol, which in turn can help lower LDL cholesterol. Lauric acid is at the core of apolipoprotein A1, which in turn is at the core of HDL cholesterol. Studies have shown higher levels of HDL particles in the blood to be very beneficial for one’s health.

I can’t recommend staying away from dairy either. As I said before, all milk fats are MCTs. If you truly are lactose intolerant, then it may be best to limit your intake to nothing more than cheese. Cheese looses its lactose as the cheese hardens, so most cheeses have little to no lactose content in them. Just don’t choose the low fat cheeses.

Butter’s Back

butterFor MCTs, I like milk fats. All milk fats are MCTs. That means that all milk fats can help you lose weight. I’ll bet you didn’t expect that, did you? That means that low fat milk and skim milk actually help make you fatter by taking away the healthy MCTs in milk fat. That also means, butter is back! Wow, how much better can it be? Butter can help you lose weight. What a concept, the more milk fat you eat, the healthier you will be. I love it!!! Cause I love cheese, and cheese is a milk fat.

MCTs are so important, Neuropharmacology just completed a study in June 2013 showing the ability of MCTs to control epileptic seizures. All MCTs are saturated fats. Your body uses saturated fats and would much rather have it fed to it than have to make its own through the ingestion of carbohydrates.

That also means that I can go back to eating bacon. I love bacon. (As a matter of fact, I’ve already gone back to eating bacon. I just enjoyed about 6 slices.) Bacon may not be a medium chain triglyceride, but it’s a saturated fat and I still love it, and I’m not restricted from eating it by my religion, so I eat it and lots of it. I couldn’t do that though, if I ate carbs. That would lead to major problems like hypertensive heart disease.

Grass fed beef is always a good source of fats as well as protein. Lamb is always good also as it’s almost always grass fed. I’m sorry vegans, but a vegetarian diet is too carbohydrate laden to be a fully healthy diet, unless you get the bulk of your calories from healthy oils like olive oil, avocado oil, palm kernel oil, and most importantly coconut oil. Simply because carbohydrates are involved in a vegetarian diet, you’re going to be suffering the same consequences as everyone else on a high carbohydrate diet. It may take longer for the disorders to manifest, because your vegan diet is a little healthier than most high carbohydrate diets, but they will, simply because carbohydrates are involved. The science of metabolism doesn’t allow any variation on this rule.

Carbs, not fats create body fat, especially visceral  fat,  the kind that kills.

The secret is to get more of your calories from fat and fewer from carbohydrates. Fat has more calories per gram of usable food anyway, making it a much more efficient fuel. Like proteins, carbohydrates only give you 4 calories per gram of food, but fat gives you 9 calories per gram of food. That’s over twice as many calories for the same weight of the food you put in your body.

That means that you have to eat more than twice as many carbohydrates to get the same amount of calories. It’s no wonder that a carbohydrate diet is so fattening.

A high fat, low carb diet is like than running high octane gas in your car but it’s even better, for your body. What high octane gas does for your car, fat does more for your body. On the other hand, What sugar does for your car, it also does to your body. Only it does it much slower.If you know of anyone who has put sugar in a gas tank, you know what that did top the engine. The same thing happens in your body. It gums it up. Glucose is to akin too glue, to be healthy.

It does it slower because your body doesn’t burn fuel as fast or as hot as your car engine, so it takes it longer to gum up. But when it does, the results are exactly the same, disastrous. That is the curse of being on a carbohydrate diet.

It’s Time For A Cure

The Value of Balancing Your Cholesterol

The Value of Balancing Your Cholesterol

cholesterol-meter-10069234Too often I hear the phrase I’ve got to get my cholesterol down. People saying this think that high cholesterol is something to fear. High cholesterol isn’t nearly as big of a problem as unbalanced cholesterol.

Cholesterol is a very important part of bodily functions and plays a major impact in your health.

To lower ones cholesterol is to endanger ones life.

Low cholesterol has been connected to depression, anxiety, bipolar disorder and statistically higher frequency of violent behavior, suicide, Parkinson’s disease, and cancer mortality. Susceptibilities to tuberculosis and gastrointestinal infections are also associated with lower cholesterol levels. Most significantly, the death rate is doubled in older adults with lower total cholesterol and stroke and cataracts rates are higher. That was according to The Great Plains Laboratory , but you can find the same message from multiple websites, proclaiming the dangers of low cholesterol.

Dr Mercola says;

The Risks of Low Cholesterol
Impaired memory and dementia are just the tip of the iceberg when it comes to low cholesterol’s impact on your brain. Having too little of this beneficial compound also:


“Unfortunately, in the United States lowering cholesterol levels has become so common that nearly everyone reading this either knows someone struggling to do so, or has struggled to do so themselves.”

Cholesterol is not the enemy

cholesterol-check-switch-10079568The Heart Association recognizes that higher HDL cholesterol levels protect against heart disease. I’ll explain how that happens later in this post. But understanding cholesterol and how it works makes it easier to understand why balancing is more important than just lowering cholesterol

“Since cholesterol is essential for all animal life, each cell synthesizes it through a complex process beginning with the mevalonate pathway and ending with a 19 step conversion of lanosterol to cholesterol. Increased dietary intake of industrial trans fats, but not ruminant saturated fats(including cholesterol), is associated with an increased risk in all-cause mortality, cardiovascular diseases or type 2 diabetes.[9]”

“Most ingested cholesterol is esterified, and esterified cholesterol is poorly absorbed. The body also compensates for any absorption of additional cholesterol by reducing cholesterol synthesis [11]”  “Biosynthesis of cholesterol is directly regulated by the cholesterol levels present, though the homeostatic mechanisms involved are only partly understood. A higher intake from food leads to a net decrease in endogenous production, whereas lower intake from food has the opposite effect.” Simply stated, the more you eat, the less you make. But because most ingested cholesterol is esterified, it’s important to know where these fats come from.

“In addition to its importance within cells, cholesterol also serves as a precursor for the biosynthesis of steroid hormones, bile acids, and vitamin D.[5] Cholesterol is crucial in the manufacture of hormones for the body’s function.  As vitamin D is crucial for brain function, cholesterol is crucial in the manufacture of vitamin. This is why statin drugs that are made for lowering cholesterol, are so dangerous.

With a substance as vital as this is, why do people want to lower it? Maybe we should look at how it floats around in your blood as it’s transported to your cells and what role that plays in the cholesterol equation.

“Cholesterol is transported inside lipoproteins.”

Cholesterol comes in many forms of lipoproteins, HDL (High Density Lipoproteins), LDL (Low Density Lipoproteins), and VLDL (Very Low Density Lipoproteins) just to name a few. It’s the HDL and LDL that we’re interested in for the sake of this post and your health. LDL and HDL are often referred to LDL cholesterol and HDL cholesterol, because that’s the type of cholesterol that makes up the respective particles. The difference in the two types is in how the cholesterol is packed in each respective particle. That tells us what kind of particles they are.  That, also, dictates how easy they are to glycate and start reeking havoc in your body. I’ll explain that after we learn the differences between these particles and it has to do with how the HDL and LDL particles are formed.

According to Wikipedia;

Low-density lipoprotein (LDL) is one of the five major groups of lipoproteins. These groups, from least dense to most dense, are: chylomicrons, very low-density lipoprotein (VLDL), intermediate-density lipoprotein (IDL), low-density lipoprotein and high-density lipoprotein (HDL), all of them, particles far smaller than human cells. In nutrition, LDL is sometimes referred to as the “bad cholesterol”.”

“Lipoproteins transfer fats around the body in the extracellular fluid, can be sampled from blood and allow fats to be taken up by the cells of the body by receptor-mediated endocytosis.[1][2] Lipoproteins are complex particles composed of multiple proteins which transport all fat molecules (lipids) around the body within the water outside cells. They are typically composed of 80-100 proteins/particle (organized by a single apolipoprotein B for LDL and the larger particles). A single LDL particle is about 260-300 nm in diameter (submicroscopic ) typically transporting 3,000 to 6,000 fat molecules/particle, varying in size according to the number and mix of fat molecules contained within. The fats carried include cholesterol, phospholipids, and triglycerides; amounts of each vary considerably.”

“LDL particles vary in size and density, and studies have shown that a pattern that has more small dense LDL particles, called Pattern B, equates to a higher risk factor for coronary heart disease (CHD) than does a pattern with more of the larger and less-dense LDL particles (Pattern A).”

“LDL particles pose a risk for cardiovascular disease when they invade the endothelium and become oxidized, since the oxidized forms are more easily retained by the proteoglycans. A complex set of biochemical reactions regulates the oxidation of LDL particles, chiefly stimulated by presence of necrotic cell debris and free radicals in the endothelium.[3] Increasing concentrations of LDL particles are strongly associated with increasing rates of accumulation of atherosclerosis within the walls of arteries over time, eventually resulting in sudden plaque ruptures and triggering clots within the artery opening, or a narrowing or closing of the opening, i.e. cardiovascular disease, stroke, and other vascular disease complications.[4]

It’s easy to see now, the importance of lowering LDL. This is what The National Library of medicine has to say about cholesterol ratios;

“Low-density lipoprotein (LDL) cholesterol concentration has been the prime index of cardiovascular disease risk and the main target for therapy. However, several lipoprotein ratios or “atherogenic indices” have been defined in an attempt to optimize the predictive capacity of the lipid profile. In this review, we summarize their pathophysiological aspects, and highlight the rationale for using these lipoprotein ratios as cardiovascular risk factors in clinical practice, specifying their cut-off risk levels and a target for lipid-lowering therapy. Total/high-density lipoprotein (HDL) cholesterol and LDL/HDL cholesterol ratios are risk indicators with greater predictive value than isolated parameters used independently, particularly LDL. Future recommendations regarding the diagnosis and treatment of dyslipidemia, including instruments for calculating cardiovascular risk or action guidelines, should include the lipoprotein ratios with greater predictive power which, in view of the evidence-based results, are none other than those which include HDL cholesterol.” With the advantages of HDL as opposed to the disadvantages of LDL, it’s become important to know the difference in HDL and LDL because a balance in the ratio seems to be more important than anything else.”

That says to me, what’s important to know is how to create HDL and how to not create LDL. This will go much farther than any medicine to balance HDL/LDL cholesterol.

Wikipedia  goes on to say;

HDL particles remove fats and cholesterol from cells, including within artery wall atheroma, and transport it back to the liver for excretion or re-utilization; thus the cholesterol carried within HDL particles (HDL-C) is sometimes called “good cholesterol” (despite being the same as cholesterol in LDL particles).”

“Increasing concentrations of HDL particles are strongly associated with decreasing accumulation of atherosclerosis within the walls of arteries. This is important because atherosclerosis eventually results in sudden plaque ruptures, cardiovascular disease, stroke and other vascular diseases. HDL particles are sometimes referred to as “good cholesterol” because they can transport fat molecules out of artery walls, reduce macrophage accumulation, and thus help prevent or even regress atherosclerosis.”

“High LDL with low HDL level is an additional risk factor for cardiovascular disease.[24]” “In a large sample of middle aged adults, low HDL cholesterol was associated with poor memory and decreasing levels over a five-year follow-up period were associated with decline in memory.[27]

With all that said from Wikipedia, it’s easy to see that not all cholesterol is equal. Some is good and some is bad. Thus is the “good cholesterol, bad cholesterol mantra”, which more than anything boasts the value of balancing your cholesterol, rather than lowering it. Knowing how to lower LDL particles while raising HDL particles would be much more beneficial than just lowering total cholesterol.

The paragraph above about HDL cholesterol says it all, increasing HDL cholesterol is a good thing, as it’sassociated with decreasing accumulation of atherosclerosis within the cell walls of the arteries”.

As you can see, HDL, the good cholesterol is something you want in your body, but the LDL,bad cholesterol is something to keep levels low in your body. Wikipedia goes on to say about HDL;

“HDL is the smallest of the lipoprotein particles. It is the densest because it contains the highest proportion of protein to lipids. Its most abundantapolipoproteins are apo A-I and apo A-II.[7] (A rare genetic variant, ApoA-1 Milano, has been documented to be far more effecitive in both protecting against and regressing arterial disease; atherosclerosis). The liver synthesizes these lipoproteins as complexes of apolipoproteins and phospholipid, which resemble cholesterol-free flattened spherical lipoprotein particles; the complexes are capable of picking up cholesterol, carried internally, from cells by interaction with the ATP-binding cassette transporter A1 (ABCA1).[8] A plasma enzyme called lecithin-cholesterol acyltransferase (LCAT) converts the free cholesterol into cholesteryl ester (a more hydrophobic form of cholesterol), which is then sequestered into the core of the lipoprotein particle, eventually causing the newly synthesized HDL to assume a spherical shape. HDL particles increase in size as they circulate through the bloodstream and incorporate more cholesterol and phospholipid molecules from cells and other lipoproteins, for example by the interaction with the ABCG1 transporter and the phospholipid transport protein (PLTP).”

“HDL transports cholesterol mostly to the liver or steroidogenic organs such as adrenals, ovary, and testes by both direct and indirect pathways. HDL is removed by HDL receptors such as scavenger receptor BI (SR-BI), which mediate the selective uptake of cholesterol from HDL. In humans, probably the most relevant pathway is the indirect one, which is mediated by cholesteryl ester transfer protein (CETP). This protein exchanges triglycerides of VLDL against cholesteryl esters of HDL. As the result, VLDLs are processed to LDL, which are removed from the circulation by the LDL receptor pathway. The triglycerides are not stable in HDL, but are degraded by hepatic lipase so that, finally, small HDL particles are left, which restart the uptake of cholesterol from cells.”

“The cholesterol delivered to the liver is excreted into the bile and, hence, intestine either directly or indirectly after conversion into bile acids. Delivery of HDL cholesterol to adrenals, ovaries, and testes is important for the synthesis of steroid hormones.”

This is why its important to get all your cholesterol into high density lipoproteins to transfer fats from cells, where they can be used to do the most good. It seems the loose floating fats, the triglycerides, VLDL and LDL cholesterol are more open, for glycation by loose glucose in the system, than the tighter more compact fats contained in the HDL packets, making them more likely to become glycated and turned into plaque.

With that said, balancing your cholesterol seems to be much more important than just lowering your cholesterol. You really don’t want to lower your good cholesterol, the HDL because of all the good it does, yet lowering the LDL with all the damage that it does, would be wise.So what is a good way to balance your cholesterol?

There are several ways to balance your cholesterol;

According to Wikipedia;

“Certain changes in diet and exercise may have a positive impact on raising HDL levels:[29]

Most saturated fats increase HDL cholesterol to varying degrees and also raise total and LDL cholesterol.[42] A high-fat, adequate-protein, low-carbohydrate ketogenic diet may have similar response to taking niacin (vitamin B3) as described below (lowered LDL and increased HDL) through beta-hydroxybutyrate coupling the Niacin receptor 1.[43]

MCTs from coconut oil increase HDL cholesterol.

“MCTs passively diffuse from the GI tract to the portal system without requirement for modification like long-chain fatty acids or very-long-chain fatty acids(longer fatty acids are absorbed into the lymphatic system). In addition, MCTs do not require bile salts for digestion. Patients who have malnutrition, malabsorption or particular fatty-acid metabolism disorders are treated with MCTs because MCTs do not require energy for absorption, use, or storage.”

“Some studies have shown that MCTs can help in the process of excess calorie burning, thus weight loss.[4][5][6][7][8] MCTs are also seen as promoting fat oxidation and reduced food intake.[9] look at all the numbers for reference, to take note all the studies done on the weight loss aspect of MCTs, there were 5 of them.

Medium Chain Triglycerides come from Coconut oil, Palm Kernel oil and dairy fats. That means that butter and cheese can actually help you lose weight and balance your cholesterol. How great is that? You can go back to eating butter with healthier consequences than eating margarine.

“Coconut milk is rich in medium-chain fatty acids (MCFAs), which the body processes differently from other saturated fats. If MCFAs are used in a diet to replace long-chain fatty acids (LCFAs) such as animal fats they may help promote weight maintenance without raising cholesterol levels.[14]

“Coconut milk contains a large proportion of lauric acid, a saturated fat that raises blood cholesterol levels by increasing the amount of high-density lipoprotein cholesterol[12]” Like coconut milk, coconut oil is high in Lauric acid.

“Medium-chain triglycerides are generally considered a good biologically inert source of energy that the human body finds reasonably easy to metabolize. They have potentially beneficial attributes in protein metabolism, but may be contraindicated in some situations due to a reported tendency to induce ketogenesis and metabolic acidosis.[12] However, there is other authority reporting no risk of ketoacidosis or ketonemia with MCTs at levels associated with normal consumption. [13]””

“Due to their ability to be absorbed rapidly by the body, medium-chain triglycerides have found use in the treatment of a variety of malabsorption ailments. MCT supplementation with a low-fat diet has been described as the cornerstone of treatment for Waldmann disease.[14] MCTs are an ingredient in some specialised parenteral nutritional emulsions in some countries (not USA).[15][16] Studies have also shown promising results for neurodegenerative disorders (e.g. Alzheimer’s and Parkinson’s diseases)[17] and epilepsy through the use of ketogenic dieting.[18][19]

“MCFA (chain lengths of 10 carbons or less are found in greatest concentrations in coconut oil, approximately 14% by weight but can also be found in butter ( approximately 9.2%) and palm kernel oil (approximately 7.2%)” “MCT oil has been taunted as a potential weight-lowering agent.”

According to the US National Library of Science, The“Weight-loss diet that includes consumption of medium-chain triacylglycerol oil leads to a greater rate of weight and fat mass loss than does olive oil2

“Thirty-one subjects completed the study (body mass index: 29.8 ± 0.4, in kg/m2). MCT oil consumption resulted in lower endpoint body weight than did olive oil (−1.67 ± 0.67 kg, unadjusted P = 0.013). There was a trend toward greater loss of fat mass (P = 0.071) and trunk fat mass (P = 0.10) with MCT consumption than with olive oil. Endpoint trunk fat mass, total fat mass, and intra-abdominal adipose tissue were all lower with MCT consumption than with olive oil consumption (all unadjusted P values < 0.05).”

So the only remaining question, is how do we lower LDL?

In my attempt to find what fats cause LDL, I’ve found nothing to suggest that eating fat causes the formation of LDL. But, on the other hand, I’ve found plenty of data that suggests,  where this kind of fat comes from. That’s is my newest post, about Apolipoprotein B. All that I’ve researched shows wheat-bread-wheat-shock-white-background-34095411that it comes from glucose. Glucose comes from starchy carbohydrates. If you want to read about how that happens, check out Carbs! The Newly Discovered Death Sentence or Diabetes Control. It all has to do with the digestion of carbs. These fats are apportioned to the visceral fat around the belly instead of fats you can use for immediate fuel. and this is where it’s formed into LDL with the help of Ribosomes from your liver. This is also where it becomes so dangerous, but you’ll have to read about it in my next post.
Again, according to Wikipedia, “Lowering the blood lipid concentration of triglycerides helps lower the concentration of small LDL particles, because fatty-acid rich VLDL particles convert in the bloodstream into small dense LDL particles.[vague]”

It makes sense then, if you want to stop the productions of LDL, you need to stop the production of triglycerides, the fuel that feeds it, and the best way is to stop that, is to curb the high starchy carbohydrates from the worst offenders, grain based foods. The guiltiest of the group is wheat, followed closely by corn, then rice and oats. All  grain based foods are at the top of this list, along with starchy vegetables like potatoes, parsnips and carrots, although carrots do have some nutritional value, like beta-carotene. All the others just don’t carry enough nutrition to counterbalance the load of carbs you get, with it.

If you’re not ready to give up your carbs, there are alternatives, to help you lower you LDL, “Niacin (B3), lowers LDL by selectively inhibiting hepatic diacylglycerol acyltransferase 2, reducing triglyceride synthesis and VLDL secretion through a receptor HM74[35] and HM74A or GPR109A.[36] “A ketogenic diet may have similar response to taking niacin (lowered LDL and increased HDL) through beta-hydroxybutyrate, a ketone body, coupling the niacin receptor (HM74A).[36]

Statin drugs are made to lower LDL also, but I can only recommend to steer clear of those, as they cause too many problems in their action of lowering LDL. As a certified caregiver, I’ve seen, too often, the ravages this drug commits the body to. They are nothing short of devastating. In every case of a patient I took care of, the patient died prematurely from the side effects of these drugs. It seems to me that in our attempt to cure ourselves, we’re killing ourselves. Cholesterol is just too important to lower.

“Because LDL particles appear harmless until they are within the blood vessel walls and oxidized by free radicals,[43] it has been postulated that ingesting antioxidants and minimizing free radical exposure may reduce LDL’s contribution to atherosclerosis, though results are not conclusive.[44][45]

I know of something far greater than ingesting antioxidants, to boost them in your system. Boosting them through calorie restriction gives you exponentially more antioxidants than eating or drinking them can ever do for you.

Because MCT ketogenic diets are made for calorie restriction and this next point deals with calorie restriction, I can see the benefits here, as well,  for added  BDNF for brain growth, increased Nrf2 for anti-oxidant production. If you don’t know about the brain growth or anti-oxidant boost of calorie restriction, check it out at the link above. I’m just beginning to understand the benefits of the MCT ketogenic diet and how much healthier it’s kept me. And if it can keep me that healthy, it can keep all those who venture to try it just as healthy.

Don’t you think it time for a cure?

I offer these to you, free!

  • MCT ketogenic diet: it can not only help you balance your cholesterol but it can help you lose weight and keep it off forever. With the proper supplements, it will help you grow your brain. Who knew that coconut oil or coconut milk could be so healthy? Who knew that butter could be so healthy? I certainly didn’t. but I do now.
  • Spices like Bay Leaf can help balance out your cholesterol as well, as described on Spices That Heal, “Research on humans showed that after one month, the bay leaf group had up to 26 % reductions in blood sugar! They also showed approximately 35 to 40% reductions in LDL cholesterol and a jump in the good HDL particles by about 25%!”

Just looking halfway through the list, I came across another half dozen spices that can all help balance cholesterol. What an excellent resource. I love free cures, since I already have these spices in my cabinet.

I have to thank Wikipedia, from which much of this post comes and is marked by quotations marks and is in italics, where used. Parts are also quoted from the National Library of Medicine and a few other independent websites. It was necessary to copy and paste the information in order to express the argument for balancing ones cholesterol, as i couldn’t have put the information in better words than it was already expressed. Again, if you find any information you feel is incorrect and have corroboration, to back up your claim, I welcome your correction to help me make this post better.