Tag Archives: vitamin D

Carbs and Arthritis

Carbs and Arthritis

Do carbs create arthritis?
Uh…..Yep!  
You Bet!!

Nothing else in the body creates inflammation, more than carbohydrates in our diet and arthritis is a disease of inflammation.

Carbs are the foundation of inflammation. They are the sole internal source of inflammation. Inflammation wouldn’t even exist  (except for external injuries) without carbohydrates.

Inflammation is caused by glucose and cholesterol coming together and glycating. It happens because of the massive amounts of glucose in the blood. Fat, by itself,  doesn’t cause inflammation. It needs glucose to do that. Protein, by itself, doesn’t cause inflammation. It needs glucose to do so, also.

That makes glucose, the bad actor in this drama, the drama of inflammation in the body and how it’s made. Every manifestation has an equation or a set amount of variables that make up that which is being manifested.

With that said, we’re going to look at the variables that make up the equation of arthritis, the variables that cause inflammation in the body, because after all, arthritis is a disease of inflammation.

Arthritis is the expression of inflammation in the body and it shows up mostly in the joints, first, where movement takes place. That’s because this is where the macrophages get deposited because this is where the blood flows.

There’s another expression of inflammation the body and it’s called a common cold. The funny thing about inflammation is that because it exists everywhere the blood flows, it affects every system in the body. A common cold, for example, expresses itself with inflammation in the sinuses. I know that this may be hard to believe, but if you remove the instigator of inflammation, carbohydrates, a common cold becomes much easier to endure. Actually, common colds are not experienced in people on a ketogenic diet nearly as much s much as they are in carbolic, those on a carbohydrate diet. This is because of the amount of inflammation that carbs cause. Viruses may play a part in the spread of a common cold, but without the glucose in the system, the expression of inflammation can’t take place.  Unfortunately, this can only be proven by elimination carbs from the diet, completely.

It’s basically the same with arthritis, because blood flows throughout the entire body and the inflammation exists in the blood, The inflammation is going to affect every system that blood flows through, including the joints of all limbs.

Before we can continue with arthritis, started we need to know what Wikipedia says about it;

“Arthritis (from Greek arthro-, joint + -itis, inflammation; plural: arthritides) is a form of joint disorder that involves inflammation in one or more joints.[1][2] There are over 100 different forms of arthritis.[3][4] The most common form of arthritis is osteoarthritis (degenerative joint disease), a result of trauma to the joint, infection of the joint, or age. Other arthritis forms are rheumatoid arthritis, psoriatic arthritis, and related autoimmune diseases. Septic arthritis is caused by joint infection.”

If the definition of arthritis is joint inflammation, We already know where inflammation comes from, and it comes from carbohydrates, as explained in Carbs, the New Death SentenceThat makes glucose the bad actor here because without the free glucose roaming through your blood, inflammation wouldn’t exist.

It’s glucose that glycates the unused proteins and fats, by attaching themselves to these cells. The glucose is looking for insulin to turn itself into fat so it can join one of the LDL particles in your blood. if it finds a protein particle or cholesterol particle (almost always LDL particles) to attach itself to, it’ll do so, and this is where the problem of inflammation begins. When this happens, the glucose glycates the cholesterol or protein and its these misshaped proteins and glycated cholesterol that forms plaque and creates inflammation.

This is where I think it gets really interesting, if the lipid that makes up the particle comes from carbohydrates, it attaches itself to an apolipoprotein B and forms LDL cholesterol to be used as fuel for the body.

If the lipid comes from fat, it associates with apolipoprotein A, the foundation of high-density particles or HDL cholesterol. Learn how the HDL and LDL work in your body by reading The value of balancing your cholesterol and The foundations of LDL cholesterol, apolipoprotein B.

It’s the LDL particles that cause most of the damage because of their loose form. Hence the name, low-density lipoprotein. These glycated particles are at the base of over half of all cancers, CVDs, brain damage and arthritis.

According to Wikipedia, “Arthritis is predominantly a disease of the elderly, but children can also be affected by the disease. More than 70% of individuals in North America affected by arthritis are over the age of 65″

This tells me that arthritis is going to happen to everyone on a carbohydrate diet, regardless of how many carbs they consume each day. Remember that 90% of the population is gluten sensitive. This is something that can only be reversed by the industry that feeds us. As long as we have to eat the food they provide us and encourage us to eat, this problem will not subside. It’s in the science, the science of inflammation.

That explains why our addiction to these vile substances must come to an end.
As a society, we need to change this pattern.

The problem of arthritis goes deeper than just inflammation, though, it rides on the amount of vitamin D in the system, as well. vitamin D is crucial to the transport of cholesterol into the cells, so it can be used.

Again, according to Wikipedia;

“Vitamin D refers to a group of fat-soluble secosteroids responsible for enhancing intestinal absorption of calcium, iron, magnesium, phosphate, and zinc. In humans, the most important compounds in this group are vitamin D3 (also known as cholecalciferol) and vitamin D2 (ergocalciferol).[1]

Vitamin D deficiency is more common in people with rheumatoid arthritis than in the general population.[36][37] However, whether vitamin D deficiency is a cause or a consequence of the disease remains unclear.[38] 1α,25-dihydroxyvitamin D3 (1,25D), an active metabolite of vitamin D, affects bone metabolism indirectly through control of calcium and phosphate homeostasis. Interaction between 1,25D and the vitamin D receptor (VDR) affects the production of RANKL and delays osteoclastogenesis.[39] Some trials have found a decreased risk for RA with vitamin D supplementation while others have not.[37]

If Rheumatoid arthritis sufferers have a deficiency of vitamin D in their bodies, that tells me that vitamin D helps to control the expression of Rheumatoid arthritis by allowing the cholesterol particle admittance into the cell so it can be used. (No conductor, no admittance.)

This action prevents the cholesterol from becoming glycated and turned into inflammation because with lower levels of vitamin D in the body, the arthritis is more prevalent. That tells me why lower levels of vitamin D increases Rheumatoid Arthritis. It’s the one-two punch that hits everyone with arthritis; carbs raise LDL particles, raising total cholesterol throwing up flags that cholesterol must be lowered. when that’s the worst thing you can do. Your cholesterol doesn’t need to be lowered (that leads to disease), it needs to be balanced, so you can continue to use your cholesterol to feed your cells the nutrients they need to function properly. See the value of balancing your cholesterol to learn how to balance yours.

Most vitamin D is produced in our skin by ultraviolet rays acting on cholesterol;

“Vitamin D3 is produced photochemically from 7-dehydrocholesterol in the skin of most vertebrate animals, including humans.[106] The precursor of vitamin D3, 7-dehydrocholesterol is produced in relatively large quantities. 7-Dehydrocholesterol reacts with UVB light at wavelengths between 270 and 300 nm, with peak synthesis occurring between 295 and 297 nm.[107] These wavelengths are present in sunlight, as well as in the light emitted by the UV lamps in tanning beds (which produce ultraviolet primarily in the UVA spectrum but typically produce 4% to 10% of the total UV emissions as UVB). Exposure to light through windows is insufficient because glass almost completely blocks UVB light.[108][109]

With vitamin D actually being a fat, in the body, as it comes from cholesterol and cholesterol is made up of lipids, that makes me wonder if it comes from digested fats or ingested fats. A look at 7-dehydrocholesterol revealed nothing as to where it comes from, so I have to be content just knowing it’s a lipid.

Being a lipid gives it access to the cellular structure of all organs, including the skin, bones, and most importantly, your brain. 

This places the importance of vitamin D even higher than what I thought before. Vitamin D is a fat that delivers calcium to your bones, making it that important to bone growth and structure. Yet it’s also important in your brain, where it acts as a conductor for cell signaling proteins, cytokines and adipokines and hormones.

According to Pubmed;
“Vitamin D receptor in the brain

It should be noted that 1,25(OH)2D signaling is conducted through the VDR, which shares its structural characteristics with the broader nuclear steroid receptor family.11 In 1992, Sutherland et al12 provided the first evidence that the VDR is expressed in the human brain. Using radiolabeled complementary deoxyribonucleic acid probes, the authors showed that VDR messenger ribonucleic acid is expressed in the postmortem brains of patients with AD or Huntington’s disease. In a landmark study, Eyles et al13 described that both the VDR and CYP27B1 are widespread in important regions of the human brain including the hippocampus, which is particularly affected by neurodegenerative disorders.1417 Furthermore, the VDR is also expressed in the prefrontal cortex, cingulate gyrus, basal forebrain, caudate/putamen, thalamus, substantia nigra, lateral geniculate nuclei, hypothalamus, and cerebellum.18 Importantly, VDR gene polymorphisms are associated with cognitive decline,19,20 AD,2124 Parkinson’s disease,2529 and multiple sclerosis.30

Showing how important vitamin D is in the brain, it’s become evident that it’s as important as the cell signaling can’t take place efficiently without it, as it’s the conductor. Without enough vitamin D in your system, the conduction is going to be poor, at best. Could it be that this is where cell degradation begins, and inflammation introduces its ugly face? Whether or not it is, we know that vitamin D is crucial for hormones and cell signaling proteins to get their signals through the cell membrane, as that’s what conductors do, they transmit signals.

That tells me, if the pathway is blocked, due to vitamin D deficiency, the cells can’t perform their intended function, because their fuel, lipoproteins can’t get through the cells, due to the lack of a conductor, vitamin D, so they’re left floating around in the blood waiting to be used.

This is where the problem begins because there’s also massive amounts of glucose floating in the blood, waiting to be turned into fat, This gives us the equation that nobody wants, Glucose + cholesterol =  glycation. Glycation is the start is the start of inflammation.

According to PubMed; “Vitamin D lipid-lowering effects appear limited to statin-treated patients and are likely due to decreased cholesterol absorption.” Cholesterol plays a much bigger part in this play than what seemed apparent a few minutes ago. If statin drugs lower total cholesterol and vitamin D, I can only imagine what damage that is reeking on the bodies of those who are condemned to use them. That tells me that those on statin drugs are condemned to more inflammation, more disease, and more arthritis, can this be true?

This is exactly why it’s so important to balance your cholesterol instead of just lowering it. The value of balancing cholesterol tells us that raising HDL cholesterol will help lower LDL cholesterol and control the inflammation by limiting the source of the inflammation, LDL cholesterol. Knowing that raising HDL particles can lower LDL particles help makes it easier to lower LDL particles. Fewer LDL particles in the blood lowers inflammation lessening the effects of arthritis in the body.

Now that we know that, We also know that lowering carb intake lowers LDL cholesterol as it’s carbs that create LDL cholesterol. So curbing carbs, even though it can’t restore, immediately, the damage that’s already been done, it can reverse its current effects, and in the future work to restore at least some of the damage. But it can only restore that which isn’t already too far damaged.

This forces me again to ask, why is this food even on our grocery shelves and why doesn’t it come with a warning?

IT’S TIME FOR A CURE!

 

The Value of Balancing Your Cholesterol

The Value of Balancing Your Cholesterol

cholesterol-meter-10069234Too often I hear the phrase I’ve got to get my cholesterol down. People saying this think that high cholesterol is something to fear. High cholesterol isn’t nearly as big of a problem as unbalanced cholesterol.

Cholesterol is a very important part of bodily functions and plays a major impact on your health.

To lower one’s cholesterol is to endanger one’s life.

Low cholesterol has been connected to depression, anxiety, bipolar disorder and statistically higher frequency of violent behavior, suicide, Parkinson’s disease, and cancer mortality. Susceptibilities to tuberculosis and gastrointestinal infections are also associated with lower cholesterol levels. Most significantly, the death rate is doubled in older adults with lower total cholesterol and stroke and cataracts rates are higher. That was according to The Great Plains Laboratory, but you can find the same message from multiple websites, proclaiming the dangers of low cholesterol.

Dr. Mercola says;

The Risks of Low Cholesterol
Impaired memory and dementia are just the tip of the iceberg when it comes to low cholesterol’s impact on your brain. Having too little of this beneficial compound also:


“Unfortunately, in the United States lowering cholesterol levels has become so common that nearly everyone reading this either knows someone struggling to do so or has struggled to do so themselves.”

Cholesterol is not the enemy

cholesterol-check-switch-10079568The Heart Association recognizes that higher HDL cholesterol levels protect against heart disease. I’ll explain how that happens later in this post. But understanding cholesterol and how it works makes it easier to understand why balancing is more important than just lowering cholesterol

“Since cholesterol is essential for all animal life, each cell synthesizes it through a complex process beginning with the mevalonate pathway and ending with a 19 step conversion of lanosterol to cholesterol. Increased dietary intake of industrial trans fats, but not ruminant saturated fats(including cholesterol), is associated with an increased risk in all-cause mortality, cardiovascular diseases or type 2 diabetes.[9]”

“Most ingested cholesterol is esterified, and esterified cholesterol is poorly absorbed. The body also compensates for any absorption of additional cholesterol by reducing cholesterol synthesis [11]”  “Biosynthesis of cholesterol is directly regulated by the cholesterol levels present, though the homeostatic mechanisms involved are only partly understood. A higher intake from food leads to a net decrease in endogenous production, whereas lower intake from food has the opposite effect.” Simply stated, the more you eat, the less you make. But because most ingested cholesterol is esterified, it’s important to know where these fats come from.

“In addition to its importance within cells, cholesterol also serves as a precursor for the biosynthesis of steroid hormones, bile acids, and vitamin D.[5] Cholesterol is crucial in the manufacture of hormones for the body’s function.  As vitamin D is crucial for brain function, cholesterol is crucial in the manufacture of vitamins. This is why statin drugs that are made for lowering cholesterol, are so dangerous.

With a substance as vital as this is, why do people want to lower it? Maybe we should look at how it floats around in your blood as it’s transported to your cells and what role that plays in the cholesterol equation.

“Cholesterol is transported inside lipoproteins.”

Cholesterol comes in many forms of lipoproteins, HDL (High-Density Lipoproteins), LDL (Low-Density Lipoproteins), and VLDL (Very-Low-Density Lipoproteins) just to name a few. It’s the HDL and LDL that we’re interested in for the sake of this post and your health. LDL and HDL are often referred to LDL cholesterol and HDL cholesterol because that’s the type of cholesterol that makes up the respective particles. The difference in the two types is in how the cholesterol is packed in each respective particle. That tells us what kind of particles they are.  That, also, dictates how easy they are to glycate and start reeking havoc in your body. I’ll explain that after we learn the differences between these particles and it has to do with how the HDL and LDL particles are formed.

According to Wikipedia;

Low-density lipoprotein (LDL) is one of the five major groups of lipoproteins. These groups, from least dense to most dense, are: chylomicrons, very low-density lipoprotein (VLDL), intermediate-density lipoprotein (IDL), low-density lipoprotein and high-density lipoprotein (HDL), all of them, particles far smaller than human cells. In nutrition, LDL is sometimes referred to as the “bad cholesterol”.”

“Lipoproteins transfer fats around the body in the extracellular fluid, can be sampled from blood and allow fats to be taken up by the cells of the body by receptor-mediated endocytosis.[1][2] Lipoproteins are complex particles composed of multiple proteins which transport all fat molecules (lipids) around the body within the water outside cells. They are typically composed of 80-100 proteins/particle (organized by a single apolipoprotein B for LDL and the larger particles). A single LDL particle is about 260-300 nm in diameter (submicroscopic ) typically transporting 3,000 to 6,000 fat molecules/particle, varying in size according to the number and mix of fat molecules contained within. The fats carried include cholesterol, phospholipids, and triglycerides; amounts of each vary considerably.”

“LDL particles vary in size and density, and studies have shown that a pattern that has more small dense LDL particles, called Pattern B, equates to a higher risk factor for coronary heart disease (CHD) than does a pattern with more of the larger and less-dense LDL particles (Pattern A).”

“LDL particles pose a risk for cardiovascular disease when they invade the endothelium and become oxidized, since the oxidized forms are more easily retained by the proteoglycans. A complex set of biochemical reactions regulates the oxidation of LDL particles, chiefly stimulated by presence of necrotic cell debris and free radicals in the endothelium.[3] Increasing concentrations of LDL particles are strongly associated with increasing rates of accumulation of atherosclerosis within the walls of arteries over time, eventually resulting in sudden plaque ruptures and triggering clots within the artery opening, or a narrowing or closing of the opening, i.e. cardiovascular disease, stroke, and other vascular disease complications.[4]

It’s easy to see now, the importance of lowering LDL. This is what The National Library of medicine has to say about cholesterol ratios;

“Low-density lipoprotein (LDL) cholesterol concentration has been the prime index of cardiovascular disease risk and the main target for therapy. However, several lipoprotein ratios or “atherogenic indices” have been defined in an attempt to optimize the predictive capacity of the lipid profile. In this review, we summarize their pathophysiological aspects, and highlight the rationale for using these lipoprotein ratios as cardiovascular risk factors in clinical practice, specifying their cut-off risk levels and a target for lipid-lowering therapy. Total/high-density lipoprotein (HDL) cholesterol and LDL/HDL cholesterol ratios are risk indicators with greater predictive value than isolated parameters used independently, particularly LDL. Future recommendations regarding the diagnosis and treatment of dyslipidemia, including instruments for calculating cardiovascular risk or action guidelines, should include the lipoprotein ratios with greater predictive power which, in view of the evidence-based results, are none other than those which include HDL cholesterol.” With the advantages of HDL as opposed to the disadvantages of LDL, it’s become important to know the difference in HDL and LDL because a balance in the ratio seems to be more important than anything else.”

That says to me, what’s important to know is how to create HDL and how to not create LDL. This will go much farther than any medicine to balance HDL/LDL cholesterol.

Wikipedia  goes on to say;

HDL particles remove fats and cholesterol from cells, including within artery wall atheroma, and transport it back to the liver for excretion or re-utilization; thus the cholesterol carried within HDL particles (HDL-C) is sometimes called “good cholesterol” (despite being the same as cholesterol in LDL particles).”

“Increasing concentrations of HDL particles are strongly associated with decreasing accumulation of atherosclerosis within the walls of arteries. This is important because atherosclerosis eventually results in sudden plaque ruptures, cardiovascular disease, stroke and other vascular diseases. HDL particles are sometimes referred to as “good cholesterol” because they can transport fat molecules out of artery walls, reduce macrophage accumulation, and thus help prevent or even regress atherosclerosis.”

“High LDL with low HDL level is an additional risk factor for cardiovascular disease.[24]” “In a large sample of middle-aged adults, low HDL cholesterol was associated with poor memory and decreasing levels over a five-year follow-up period were associated with decline in memory.[27]

With all that said from Wikipedia, it’s easy to see that not all cholesterol is equal. Some are good and some are bad. Thus, the “good cholesterol, bad cholesterol mantra”, which more than anything boasts the value of balancing your cholesterol, rather than lowering it. Knowing how to lower LDL particles while raising HDL particles would be much more beneficial than just lowering total cholesterol.

The paragraph above about HDL cholesterol says it all, increasing HDL cholesterol is a good thing, as it’sassociated with decreasing accumulation of atherosclerosis within the cell walls of the arteries”.

As you can see, HDL, the good cholesterol is something you want in your body, but the LDL, bad cholesterol is something to keep levels low in your body. Wikipedia goes on to say about HDL;

“HDL is the smallest of the lipoprotein particles. It is the densest because it contains the highest proportion of protein to lipids. Its most abundant apolipoproteins are apo A-I and apo A-II.[7] (A rare genetic variant, ApoA-1 Milano, has been documented to be far more effecitive in both protecting against and regressing arterial disease; atherosclerosis). The liver synthesizes these lipoproteins as complexes of apolipoproteins and phospholipid, which resemble cholesterol-free flattened spherical lipoprotein particles; the complexes are capable of picking up cholesterol, carried internally, from cells by interaction with the ATP-binding cassette transporter A1 (ABCA1).[8] A plasma enzyme called lecithin-cholesterol acyltransferase (LCAT) converts the free cholesterol into cholesteryl ester (a more hydrophobic form of cholesterol), which is then sequestered into the core of the lipoprotein particle, eventually causing the newly synthesized HDL to assume a spherical shape. HDL particles increase in size as they circulate through the bloodstream and incorporate more cholesterol and phospholipid molecules from cells and other lipoproteins, for example by the interaction with the ABCG1 transporter and the phospholipid transport protein (PLTP).”

“HDL transports cholesterol mostly to the liver or steroidogenic organs such as adrenals, ovary, and testes by both direct and indirect pathways. HDL is removed by HDL receptors such as scavenger receptor BI (SR-BI), which mediate the selective uptake of cholesterol from HDL. In humans, probably the most relevant pathway is the indirect one, which is mediated by cholesteryl ester transfer protein (CETP). This protein exchanges triglycerides of VLDL against cholesteryl esters of HDL. As the result, VLDLs are processed to LDL, which are removed from the circulation by the LDL receptor pathway. The triglycerides are not stable in HDL, but are degraded by hepatic lipase so that, finally, small HDL particles are left, which restart the uptake of cholesterol from cells.”

“The cholesterol delivered to the liver is excreted into the bile and, hence, intestine either directly or indirectly after conversion into bile acids. Delivery of HDL cholesterol to adrenals, ovaries, and testes is important for the synthesis of steroid hormones.”

This is why it’s important to get your cholesterol into high-density lipoproteins to transfer fats from cells, where they can be used to do the most good. It seems the loose floating fats, the triglycerides, VLDL and LDL cholesterol are more open, for glycation by loose glucose in the system than the tighter more compact fats contained in the HDL packets, making them more likely to become glycated and turned into plaque.

With that said, balancing your cholesterol seems to be much more important than just lowering your cholesterol. You really don’t want to lower your good cholesterol, the HDL because of all the good it does, yet lowering the LDL with all the damage that it does, would be wise.So what is a good way to balance your cholesterol?

There are several ways to balance your cholesterol;

According to Wikipedia;

“Certain changes in diet and exercise may have a positive impact on raising HDL levels:[29]

Most saturated fats increase HDL cholesterol to varying degrees and also raise total and LDL cholesterol.[42] A high-fat, adequate-protein, low-carbohydrate ketogenic diet may have similar response to taking niacin (vitamin B3) as described below (lowered LDL and increased HDL) through beta-hydroxybutyrate coupling the Niacin receptor 1.[43]

MCTs from coconut oil increase HDL cholesterol.

“MCTs passively diffuse from the GI tract to the portal system without requirement for modification like long-chain fatty acids or very-long-chain fatty acids(longer fatty acids are absorbed into the lymphatic system). In addition, MCTs do not require bile salts for digestion. Patients who have malnutrition, malabsorption or particular fatty-acid metabolism disorders are treated with MCTs because MCTs do not require energy for absorption, use, or storage.”

“Some studies have shown that MCTs can help in the process of excess calorie burning, thus weight loss.[4][5][6][7][8] MCTs are also seen as promoting fat oxidation and reduced food intake.[9] look at all the numbers for reference, to take note all the studies done on the weight loss aspect of MCTs, there were 5 of them.

Medium Chain Triglycerides come from Coconut oil, Palm Kernel oil and dairy fats. That means that butter and cheese can actually help you lose weight and balance your cholesterol. How great is that? You can go back to eating butter with healthier consequences than eating margarine.

“Coconut milk is rich in medium-chain fatty acids (MCFAs), which the body processes differently from other saturated fats. If MCFAs are used in a diet to replace long-chain fatty acids (LCFAs) such as animal fats they may help promote weight maintenance without raising cholesterol levels.[14]

“Coconut milk contains a large proportion of lauric acid, a saturated fat that raises blood cholesterol levels by increasing the amount of high-density lipoprotein cholesterol[12]” Like coconut milk, coconut oil is high in Lauric acid.

“Medium-chain triglycerides are generally considered a good biologically inert source of energy that the human body finds reasonably easy to metabolize. They have potentially beneficial attributes in protein metabolism, but may be contraindicated in some situations due to a reported tendency to induce ketogenesis and metabolic acidosis.[12] However, there is other authority reporting no risk of ketoacidosis or ketonemia with MCTs at levels associated with normal consumption. [13]””

“Due to their ability to be absorbed rapidly by the body, medium-chain triglycerides have found use in the treatment of a variety of malabsorption ailments. MCT supplementation with a low-fat diet has been described as the cornerstone of treatment for Waldmann disease.[14] MCTs are an ingredient in some specialised parenteral nutritional emulsions in some countries (not USA).[15][16] Studies have also shown promising results for neurodegenerative disorders (e.g. Alzheimer’s and Parkinson’s diseases)[17] and epilepsy through the use of ketogenic dieting.[18][19]

“MCFA (chain lengths of 10 carbons or less are found in greatest concentrations in coconut oil, approximately 14% by weight but can also be found in butter ( approximately 9.2%) and palm kernel oil (approximately 7.2%)” “MCT oil has been taunted as a potential weight-lowering agent.”

According to the US National Library of Science, The“Weight-loss diet that includes consumption of medium-chain triacylglycerol oil leads to a greater rate of weight and fat mass loss than does olive oil2

“Thirty-one subjects completed the study (body mass index: 29.8 ± 0.4, in kg/m2). MCT oil consumption resulted in lower endpoint body weight than did olive oil (−1.67 ± 0.67 kg, unadjusted P = 0.013). There was a trend toward greater loss of fat mass (P = 0.071) and trunk fat mass (P = 0.10) with MCT consumption than with olive oil. Endpoint trunk fat mass, total fat mass, and intra-abdominal adipose tissue were all lower with MCT consumption than with olive oil consumption (all unadjusted P values < 0.05).”

So the only remaining question, is how do we lower LDL?

In my attempt to find what fats cause LDL, I’ve found nothing to suggest that eating fat causes the formation of LDL. But, on the other hand, I’ve found plenty of data that suggests,  where this kind of fat comes from. That’s is my newest post, about Apolipoprotein B. All that I’ve researched shows wheat-bread-wheat-shock-white-background-34095411that it comes from glucose. Glucose comes from starchy carbohydrates. If you want to read about how that happens, check out Carbs! The Newly Discovered Death Sentence or Diabetes Control. It all has to do with the digestion of carbs. These fats are apportioned to the visceral fat around the belly instead of fats you can use for immediate fuel. and this is where it’s formed into LDL with the help of Ribosomes from your liver. This is also where it becomes so dangerous, but you’ll have to read about it in my next post.
Again, according to Wikipedia, “Lowering the blood lipid concentration of triglycerides helps lower the concentration of small LDL particles because fatty-acid rich VLDL particles convert in the bloodstream into small dense LDL particles.[vague]”

It makes sense then, if you want to stop the productions of LDL, you need to stop the production of triglycerides, the fuel that feeds it, and the best way is to stop that, is to curb the high starchy carbohydrates from the worst offenders, grain-based foods. The guiltiest of the group is wheat, followed closely by corn, then rice and oats. All grain-based foods are at the top of this list, along with starchy vegetables like potatoes, parsnips and carrots, although carrots do have some nutritional value, like beta-carotene. All the others just don’t carry enough nutrition to counterbalance the load of carbs you get, with it.

If you’re not ready to give up your carbs, there are alternatives, to help you lower your LDL, “Niacin (B3), lowers LDL by selectively inhibiting hepatic diacylglycerol acyltransferase 2, reducing triglyceride synthesis and VLDL secretion through a receptor HM74[35] and HM74A or GPR109A.[36] “A ketogenic diet may have similar response to taking niacin (lowered LDL and increased HDL) through beta-hydroxybutyrate, a ketone body, coupling the niacin receptor (HM74A).[36]

Statin drugs are made to lower LDL also, but I can only recommend steering clear of those, as they cause too many problems in their action of lowering LDL. As a certified caregiver, I’ve seen, too often, the ravages this drug commits to the body. They are nothing short of devastating. In every case of a patient I took care of, the patient died prematurely from the side effects of these drugs. It seems to me that in our attempt to cure ourselves, we’re killing ourselves. Cholesterol is just too important to lower.

“Because LDL particles appear harmless until they are within the blood vessel walls and oxidized by free radicals,[43] it has been postulated that ingesting antioxidants and minimizing free radical exposure may reduce LDL’s contribution to atherosclerosis, though results are not conclusive.[44][45]

I know of something far greater than ingesting antioxidants, to boost them in your system. Boosting them through calorie restriction gives you exponentially more antioxidants than eating or drinking them can ever do for you.

Because MCT ketogenic diets are made for calorie restriction and this next point deals with calorie restriction, I can see the benefits here, as well,  for added  BDNF for brain growth, increased Nrf2 for anti-oxidant production. If you don’t know about the brain growth or anti-oxidant boost of calorie restriction, check it out at the link above. I’m just beginning to understand the benefits of the MCT ketogenic diet and how much healthier it’s kept me. And if it can keep me that healthy, it can keep all those who venture to try it just as healthy.

Don’t you think it’s time for a cure?

I offer these to you, free!

  • MCT ketogenic diet: it can not only help you balance your cholesterol but it can help you lose weight and keep it off forever. With the proper supplements, it will help you grow your brain. Who knew that coconut oil or coconut milk could be so healthy? Who knew that butter could be so healthy? I certainly didn’t. but I do now.
  • Spices like Bay Leaf can help balance out your cholesterol as well, as described on Spices That Heal, “Research on humans showed that after one month, the bay leaf group had up to 26 % reductions in blood sugar! They also showed approximately 35 to 40% reductions in LDL cholesterol and a jump in the good HDL particles by about 25%!”

Just looking halfway through the list, I came across another half dozen spices that can all help balance cholesterol. What an excellent resource. I love free cures since I already have these spices in my cabinet.

I have to thank Wikipedia, from which much of this post comes and is marked by quotations marks and is in italics, where used. Parts are also quoted from the National Library of Medicine and a few other independent websites. It was necessary to copy and paste the information in order to express the argument for balancing one’s cholesterol, as I couldn’t have put the information in better words than it was already expressed. Again, if you find any information you feel is incorrect and have corroboration, to back up your claim, I welcome your correction to help me make this post better.